BISNU PRASAD DASH
Reader
M.Sc. -1983 (Utkal University);
M.Phil. 1986; Ph.D. - 1994 (Sambalpur Univ.)
Animal Physiology
and Biochemistry, Haematology
Research Experience: Sixteen
years at RMRC, ICMR, Bhubaneswar
Teaching Experience: Three
years
Publications: 29 (International
3, National 26)
Referee: Annals of Haematology
(International Journal)
Research summary:
· Red blood cell glucose metabolism with special emphasis
on sickle red cell .
· Distribution of abnormal haemoglobins in different caste
groups of Orissa.
· Biology of sickle cell diseases, Beta thalassaemia, and
double heterozygotes for abnormal haemoglobins in Orissa.
· Prevalence of sickle cell hemoglobin ,Beta thalassaemia,
G6PD deficiency in fifteen major tribes of Orissa.
· Rare case of sickle cell patient with Priapism
· Occurrence of Haemoglobin E beta thalassaemia in Orissa.
· P. falciparum genotypes with reference to severe malaria
in Orissa.
Selected papers
1. Ranjit, M.,R, Das, A, Das, B.P, Das, B. N, Dash, B. P and Chhotray,G.P.(2005)
Distribution of P. falciparum genotypes in clinically mild and
severe malaria cases in Orissa, India. Trans. Royal. Soc. Trop.
Med & Hyg. 99: 389-395.
2. Balgir, R.S., Dash, B..P and Murmu, B.(2004) Blood groups,hemoglobinopathy
and G6PD deficiency investigations among fifteen major tribes
of Orissa,India.Anthropologist,6(1): 69-75.
3. Chhotray, G.P., Dash, B.P. and Ranjit ,M.R.(2004) Spectrum
of hemoglobinopathies in Orissa,India. Hemoglobin. 28(2): 117-122.
4. Chhotray, G.P., Dash, B.P., Ranjit, M.R., Cohln, R. and Mohanty,
D.(2003) Hemoglobin E beta thalassaemia- An experience in eastwrn
Indian state of Orissa. Acta Haematologica 109: 212-214.
5. Dash, B.P., Mittra, A. and Kar, B.C. (1999) Osmotic fragility
of normal and sickle haemoglobin containing red blood cells. Indian
J Physiol Pharmacol. 43(2): 267-269.
6. Mohapatra, B.N., Dash,B.P. and Kar, B.C.(1993) Serum Immunoglobulins
in sickle cell disease. JAPI, 41: 418-419.
7. Dash, B.P., Mittra, A and Kar, B.C. (1992) A study on the glucose
uptake, pyruvate and lactate formation in red blood cells of normal,
sickle cell trait and sickle cell patients. Indian J Clin Biochem,
7(2): 134-137.